Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesis.



Liang, Bo and Soka, Magdalena and Christensen, Alex Horby and Olesen, Morten S and Larsen, Anders P and Knop, Filip K and Wang, Fan and Nielsen, Jonas B and Andersen, Martin N and Humphreys, David T and Mann, Stefan A and Huttner, Inken G and Vandenberg, Jamie I and Svendsen, Jesper H and Haunsø, Stig and Preiss, Thomas and Seebohm, Guiscard and Olesen, Søren-Peter and Schmitt, Nicole and Fatkin, Diane (2014) Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesis. Journal of Molecular and Cellular Cardiology, 67. pp.69-76. ISSN 1095-8584 (PP OA)

Link to published document: http://dx.doi.org/10.1016/j.yjmcc.2013.12.014

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Subjects: R Medicine > R Medicine (General)
Depositing User: Repository Administrator
Date Deposited: 11 Dec 2015 00:06
Last Modified: 23 Jan 2018 02:42

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Filename: Liang 2014 Task1 channel_JMCC PP.pdf